By Anish Bhardwaj, Jeffrey R. Kirsch

ISBN-10: 1420047949

ISBN-13: 9781420047943

The scientific administration of sufferers with acute mind and spinal wire damage has developed considerably with the arrival of recent diagnostic and healing modalities. Editors Bhardwaj, Ellegala, and Kirsch current Acute mind and Spinal twine Injury , a brand new stand-alone connection with support today’s neurologists and neurosurgeons hold abreast of the entire contemporary developments in mind and spinal wire damage. Divided into 5 sections, demanding mind harm, ischemic stroke, intracerebral and subarachnoid hemorrhage, and spinal wire harm, this article bargains the most up-tp-date scientific technology and highlights controversies within the scientific administration of sufferers with acute mind and spinal twine injuries.

Acute mind and Spinal twine Injury :

  • each part delineates diagnostic and tracking instruments, pharmacotherapies, and interventional and surgical remedies are covered
  • examines and explores lately released laboratory trials and research
  • incorporates over 50 diagrams and figures for concise communique of clinical information

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The scientific administration of sufferers with acute mind and spinal wire harm has advanced considerably with the appearance of recent diagnostic and healing modalities. Editors Bhardwaj, Ellegala, and Kirsch current Acute mind and Spinal wire harm , a brand new stand-alone connection with support today’s neurologists and neurosurgeons retain abreast of the entire contemporary developments in mind and spinal twine damage.

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Extra resources for Acute Brain and Spinal Cord Injury: Evolving Paradigms and Management

Example text

Elevated Intracranial Pressure Nearly 50% of head-injured patients with intracranial mass lesions and 33% of patients with diffuse axonal injury have persistently elevated ICP. Increased ICP also compromises cerebral perfusion. Aside from the fact that increased ICP can decrease cerebral perfusion pressure (CPP), it is also true that drops in CPP can increase ICP. As CPP decreases, pial arterioles vasodilate and accommodate larger CBV, diagrammed in Rosner’s vasodilatory cascade (23). This increased Decompressive Hemicraniectomy 7 CBV can increase ICP.

Three types of secondary cranial decompression are generally discussed: unilateral, bilateral, and temporal decompression. No experimentally validated criteria exist to guide neurosurgeons in their choice of procedure, but some empirically derived conclusions may be drawn. In patients with medically refractory ICP with a largely unilateral TBI, a large one-sided frontotemporoparietal decompression is often considered; an absolutely critical element of this procedure is adequate temporal release, confirmed by reaching the floor of the middle fossa at surgery and confirming adequate anterior temporal bony decompression (Fig.

Crit Care Med 2003; 31 (10):2535–2538. 41. Csokay A, Egyud L, Nagy L, et al. Vascular tunnel creation to improve the efficacy of decompressive craniotomy in post-traumatic cerebral edema and ischemic stroke. Surg Neurol 2002; 57(2):126–129. 42. Kontopoulos V, Foroglou N, Patsalas J, et al. Decompressive craniectomy for the management of patients with refractory hypertension: should it be reconsidered? Acta Neurochir (Wien) 2002; 144(8):791–796. 43. Skoglund TS, Eriksson-Ritzen C, Jensen C, et al.

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Acute Brain and Spinal Cord Injury: Evolving Paradigms and Management by Anish Bhardwaj, Jeffrey R. Kirsch


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